Like an Enquirer reader, I want to know.
This link gave some short definitions:
http://users.rcn.com/jkimball.ma.ultranet/BiologyPages/M/Memory.html
Be aware that while long term memories will survive a number of events that will disrupt short term memories (electrical shocks, anoxia, severe blood loss, concussions, some forms of chemical intoxication, etc.), this does not mean that they cannot be lost.
One critical distinction is between "Implicit" long term memory--which is well understood and involves alterations in the synaptic patterns of the brain--and "Explicit" memories--which are a total mystery. "Implicit" memories cantheoretically be "overwritten" and lost (particularly by experiences that involve prolonged stress). If "Explicit" memories are stored in the synaptic patterns on the same basic principle, then they too can be "overwritten" (research seems to indicate that--whether or not "Explicit" memories are stored in synaptic patterns or not--they can become overwritten by false memories).
Which makes it a question of definition. Is a brain in which "actual" memories have become overwritten by "false" memories "intact and uninjured," or is it functionally damaged?
http://www.enidreed.com/serv01.htm
I'm not sure what this means in terms of memory, but if people can function well, and even excel, on *that* little brain tissue... <sighs> I'm obviously not making full use of my gray matter...
In the case of hydrocephalic tissue damage, I can only offer the postulate that the number of synaptic connections per neuron increases as surviving neurons slowly comphensate for the lost neurons. Or perhaps I should say, fails to decrease. In infants, there are far more synaptic connections per neuron than in adults, and during the maturation process unused synapses are lost. Whether a hydrocephalic person suffers impairment may have almost everything to do with whether tissue damage occurs prior to this "culling" process.
Which explains almost nothing, of course.
Memories are contained in the human body, which is reliant on an oxygen supply to stay alive. When a memory isn't called upon, it becomes less and less needed, restricting the oxygen flow to it, tranferring it's wasted potential to more prevalent memories (or other places in the body). Once something (including memories no doubt) is increasingly suffocated, it dies off.
Although in the case of memories, the tissue itself (probably?) wouldn't die, the memory would be lost.
Results: Subjects relearned the original associations faster (in spite of the fact that they could not recall them). Subjects relearned the original associations faster (in spite of the fact that they could not recall them). This suggests that some associative information was retained. One possible interpretation: strength of memories decay gradually. If these strengths fall below a certain threshold, we can't recall the information, but the remaining memory trace is still there to facilitate relearning."
Anyway, I guess what the instructors say about practice making perfect is true.
I'm not sure I understand what DevOp is trying to say about memories, though.
DevOp, I still can't understand what you're saying. Rephrase that using the definitions found in tamalynn's link, if you would.
I.e., traumatic memories can be triggered by a smell, a sound, a sight, a sensation - and these tramautic memories, for the person remembering, seem to be akin to the experience the first time around.
Good/pleasant memories can be vividly brought to life through association, also - although it doesn't seem to occur as much as tramautic memories.
Perhaps that has to do with the chemical imbalance created by trauma throughout the body, when the person is unable to "come down" off the adrenaline high that happened as a protective, life saving measure.
Have there been any studies on "chemical" memory? Or is that just a way out there, silly notion?